MMS works, effective against infectious microorganisms and viruses is described with emphasis on Chlorine Dioxide (CLO2

Introduction
CLO2 is an inorganic compound composed of oxygen; chloride; potassium and gold. Compounds of oxygen and chloride are formed from two electronegative elements. This chemical property of CLO2 makes possible the release of nascent oxygen upon decomposition during its action as an anti-microbial agent. Certain aspects of the cellular immune system (specifically white blood cells) utilize other mechanisms in the generation of highly reactive oxygen derivatives (free radicals) for the purpose of combating the invasion of foreign organisms. Without these protective mechanisms provided by the immune system involving oxygen derivatives; the ability to fight infection is greatly impaired.

The immune system of many persons; particularly the elderly; is deficient in the ability of providing these highly reactive oxygen derivatives (free-radicals) which are so necessary for attacking the great variety of viral; and bacterial invaders become easy targets for the many diseases they produce with the accompanying and sometimes bizarre symptoms.

The use of CLO2 assists the natural protective mechanisms of the body in counteracting these infectious agents which; if not adequately neutralized; will most certainly lead to disease. There will be described below the biochemical mechanisms by which CLO2 assists the immune system in destroying pathogenic organisms by making available an active form of oxygen [1].

Antiviral activity of CLO2

Viruses; in general; consist of two parts; an inner nucleic acid core consisting of either; DNA or RNA; and an outer protein coat. The protein coat not only serves to protect the vital nucleic acid within but is also instrumental in providing specificity in binding to the surface of particular cells (polio virus binds to nerve cells; influenza virus binds to the nasal mucosa; etc.). It has also been shown that without certain specific polysaccharides attached to the protein coat (glycoproteins); binding to the cell surface will not occur [30].

This aspect will be discussed in greater detail below in relation to the action of CLO2 on viruses.

Some virus; including the AIDS virus HTLV-I3I; herpes and cytomegalovirus; are surrounded by a bilayer lipid membrane which is a fragment of the plasma membrane of the cell in which the virus replicated. As the virus ruptures and kills the cell it takes with it a fragment of the plasma membrane of that cell and becomes encapsulated. This membrane is critical for infectivity and without it the virus cannot enter the proper cell type for replication. Anti-viral agents which damage or deteriorate the encapsulating bilayer lipid membrane also inhibit or prevent infectivity. We shall return to this aspect of the anti-viral activity of CLO2 in a subsequent section.

Biochemical activity of CL02

The action of CLO2 as a cytotoxic agent may include as many as four separate biochemical mechanisms described below.

Oxidation of protein

As mentioned above; the protein coat is highly significant in viral infectivity and provides specificity for binding to the proper cell type. Two amino acids that would undergo oxidation and modification to a significant enough degree to permit inhibition of virus to cell binding are those containing sulfur; namely; methionine and cysteine.

Note corono virus has a bilayer lipid membrane as described above, which chlorine dioxide destorys.

Virus inactivation

A virus typically consists of an outer shell or coating of protein encapsulating a nucleic acid which may be either DNA or RNA (e.g. Retroviruses). The skeletal backbone of nucleic acids includes derivatives of phosphoric acid (H3PO4; a very strong acid) in which two of the original three hydroxyl groups (-OH) are substituted; leaving only one active hydroxyl group per phosphate.

Segment of extended dna chain showing alternating deoxyribose- phosphate linkage

Some viruses may have glycoproteins incorporated into their protein coat; that is; proteins to which polysaccharides (sugar chains) have been attached. The bound polysaccharides may attach to specific sites on the protein coat; effectively converting a protein surface into a poly- sugar surface [3]. This new surface has specificity for certain polysaccharides found on the surfaces of specific cell types; thereby conveying specificity to virus binding as well as a degree of immunological protection.

Once bound to the appropriate cell type; the nucleic acid component of the virus is injected into the cell and in many ways; takes over the protein synthesis processes of the cell. Certain segments of the viral nucleic acid consist of genes that are responsible for the replication of the coat. The nucleic acid component replicates by a process known as “base-pairing”; in which each base of the original strand attracts and binds the corresponding base; forming a pair (A-T and C-G; described more fully below) through hydrogen bonding. The result is the replication of the complete virus until the cell bursts; releasing many additional viral particles into the surrounding medium. In the presence of these acidic nucleic acids; the CLO2 molecule becomes unstable and releases nascent oxygen into the medium.

Nucleic acids; both RNA and DNA; have many characteristics in common; in fact; they are almost chemically identical. Each consists of a chain of alternating sugar (a modification of ribose; deoxyribose) and phosphate groups; known as the “phosphate-sugar backbone”. Attached to a specific carbon of each deoxyribose group is an organic ring compound known as a “base”. Altogether; there are only four types of bases found in DNA; namely; guanine (G); cytosine (C); adenine (A) and thymine (T). It is the sequence of these four units along the chain that makes one segment of DNA differs from another. RNA differs chemically from DNA in that the base thymine is replaced by the base uracil (U); representing a very subtle biochemical difference. There is also a subtle difference in the sugar deoxyribose.

The release of nascent oxygen from chlorine dioxide CLO2 in an acidic environment

The base guanine; found in both RNA and DNA; is very sensitive to oxidation; forming 8-oxoguanine as the oxidation product [4]. The release of CLO2 results in the oxidation of the guanine residue with the formation of 8-oxoguanine; thereby disallowing the replication of the viral nucleic acid by base pairing. Although the replication of the protein coat may continue; the formation of a complete functional virus has been blocked by CLO2 oxidation.

Fate of CLO2 following ingestion

The half-life for the elimination of CLO2 from experimental animals (rat) was 43.9hours. When rats drinking 100mg of CLO2 per liter for 15days were then given 3ml of 300mg/liter orally the half-life was 31hours.

The distribution of CLO2 in tissues following administration of CLO2 labelled material is as follows

Plasma; 2.74; Kidney; 2.45; Lung; 2.25; Stomach; 2.15; Liver; 1.16; Spleen; 0.76; Thymus; 0.68 [20]. The metabolites of CLO2 are chlorite (CLO2); chlorate (CIO3); chloride (CL) and oxygen (O2) [21,22] (Figure 5).

Toxicity of CLO2

It has been shown by toxicity studies in man that daily ingestion of 500ml CLO2 having a concentration of 5ppm is safely tolerated [23]. In other studies with rats the LD of NaCLO2 was established as 140mg/kg [2]. In studies where CLO2; Na CLO2 or CIO3 was included in the drinking water for up to several months where no significant increases in methemoglobin concentrations resulted with doses as high as 1000mg/liter (1000ppm) in the rat; mouse or chicken [20].

Clinical application of CLO2

Ongoing research at the Innerlight Biological Research Foundation has been investigating the clinical usage of CLO2 for the following applications for over twenty-five years.

Herpes I and HI
AIDS (HTLV-m)[45]
Sty (eye)
Cold sores (herpes)
Shingles (herpes)
Warts
Cytomegalovirus
Polio
Influenza
Canker sore
Ear infection
Chronic bronchitis Sinusitis
Dandruff
Psoriasis
Strep throat
Pseudomonas Pneumonia
Peridontal disease
Bladder infections
Mycotic (Fungal)
Candida (systemic)
Candida (snails)[45]
Candida (vaginitis)
Candida (oral; thrush)
Candida (gastrointestinal)
Ringworm Scleroderma
This research also includes nonspecific spores; bacteria and virus.

Clinical outcome studies of CLO2

Extensive clinical applications of CLO2 to Epstein-Barr virus (EBV) [46]; cytomegalovirus (CMV); hepatitis virus A; B; HIV (AIDS virus) and others are being used continually. The DNA of EBV within the virus itself is in a linear form. Sometime after infection; the ends are linked together; forming the circular form (episome). Once this form of DNA is firmly established; the cell is said to be in a latent state. The virus remains in this state in certain B-cells for the remainder of the patient’s life. About 10% of the B-cells are in the actively proliferating state [47]. An Epstein-Barr clinical study was conducted in the American Biologics Medical Center over a four year period from 1992 to 1996 [46].

Case load:There were 1207 patients treated with the Dioxychlor® protocol. 784 patients were female - 65%. 423 patients were male - 35%. Ages ranged from 16 to 52 years.

Good work OP. Get you case of beer for that one.

thanks, make it a corona. lol

How do you make it OP?

Good stuff! Thank you

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Thank you!

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MMS saved my life twice and there is no human being on earth who will convince me not to use it.

Great stuff, cured me of dengue twice within 3 hrs each time. Care to share your experience in more depth

hello. i'm trying to find out the correct mixture to make mms with oxine & citris acid. i have some info but not complete & trusted. also is mms a cure for covid or a preventative measure ? thanks

This is fascinating and I have just begun taking the starting protocol. What do people think the #s of drops per day to fight corona are?

*BUMP*

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